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Some eicosanoids exacerbate AD pathology, while others exhibit beneficial effects (Biringer 2019)

Collectively, these multifaceted disruptions lead to persistent hyperarousal, characterized by an imbalance between arousal-promoting neuronal circuits (e.g., the locus coeruleus and hypothalamic orexin neurons) and sleep-promoting circuits (e.g., the VLPO and basal forebrain)

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