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By 96 hours, mucus flow diminished, with 75% showing some discharge and 25% none

A homozygous R148W mutation in semaphorin 7A causes progressive familial intrahepatic cholestasis

Its working mechanism is allosteric facilitation of HGF/c-Met signaling: Dihexa binds HGF with high affinity (reported K d ~65 pM in the original work) and is proposed to potentiate HGF dimerization and c-Met phosphorylation at subthreshold endogenous HGF concentrations

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Lastly, the identification of an imbalance in the redox environment was recognised because of the upregulation of glutathione metabolism (Fig

Persky AM, Brazeau GA, Hochhaus G