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When excessive glutamate is released from the presynaptic, it stimulates the postsynaptic ionotropic glutamate receptors (iGluRs) and metabotropic glutamate receptors (mGluRs)

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Longitudinal studies in adults with sepsis have shown that glucocorticoid dysregulation begins with an initial rise in free cortisol, decreased hepatic CBG production, and ACTHcortisol dissociation during the acute phase (312, 313)

You might have heard murmurs about some new glutathione rush flooding social media but is this antioxidant asset safe or just another potentially problematic fad