slc25a39 glutathione overexpression exacerbates lung adenocarcinoma progression and is negatively regulated by AFG3L2 DLAT sustains redox homeostasis and
DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39 mediated mitochondrial glutathione transport ScienceDirect Slc25a39 and Slc25a40 Expression in Mice with Bile Duct Ligation or Lipopolysaccharide Treatment SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Nature SLC25A39 Fusion Protein Ag7141 Proteintech mitochondrial glutathione transporter import identified as key driver of breast cancer spread Mitochondrial glutathione, imported via slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation
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