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We found that postnatal vB12 treatment corrected some of the metabolic alterations and it eradicated the PPI deficits

Excessive ROS production leads to detrimental effects on mitochondrial proteins and lipids, causing oxidative damage and facilitating ferroptosis ( The underlying cause of ferroptosis is a disequilibrium between the bodys oxidants and antioxidants

there were duplicate publications of the study, and only the version with the most complete data was retained

Neurotransmitter modulation: acetyl-L-carnitine donates acetyl groups for acetylcholine synthesis and epigenetic regulation of BDNF and mGlu2 expression

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Gas6 deficiency increases oligodendrocyte loss and microglial activation in response to cuprizone-induced demyelination