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Phospholipid hydroperoxide GPx4 (PH-GPx) expressed in most tissues, can reduce many hydroperoxides including hydroperoxides integrated in membranes, hydroperoxy lipids in low density lipoprotein or thymine

Under pathologic conditions and in the presence of transforming growth factor beta (TGF-), fibroblasts accumulate in these areas of damage and differentiate into myofibroblasts that secrete collagen and other proteins

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Therefore, the present review aims to systematically dissect the core molecular mechanisms of ferroptosis, elucidate its specific pathogenic roles in cognitive disorders including AD, PD and DACI as well as provide a comprehensive overview of emerging diagnostic biomarkers and therapeutic strategies targeting this cell death pathway

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Inclusion Criteria: Priority was given to primary original articles with higher empirical values, evidence-based methodology, narrative and systematic reviews in English Language only