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Moreover, the rupture of the blood vessel wall in hemorrhagic stroke results in the accumulation of hemoglobin and heme iron at the extracellular milieu and subsequent iron-induced LPO and ferroptosis, thereby furtherly exacerbating the stroke-induced brain injury

NO produced by UV-exposed keratinocytes stimulates the -MSH/MC1R pathway, activating tyrosinase, the key enzyme in melanin synthesis

Dexmedetomidine increases lipid peroxidation levels in the diaphragm of mechanically ventilated rats

Retention of LDL in the intima, due to interactions with extracellular matrix such as chondroitin sulfate-rich proteoglycans, sequesters LDL away from the antioxidant environment of the plasma and exposes LDL to oxidation

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