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These GIs are involved in meningococcal adhesion and subsequent host cell invasion, iron uptake ( hpuAB ), meningococcal survival in the intracellular environment, modulation of the host cell cycle, innate immune escape and evasion of phagocytic killing, epigenetic control of gene expression ( modB ), and competition within the meningococcal population in the human nasopharynx

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slund F, Zheng M, Beckwith J, Storz G

[DOI] [PubMed] [Google Scholar] 82.Latha R.C.R., Daisy P

Sniegowski T, Korac K, Bhutia YD, Ganapathy V