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Abstract Traumatic brain injury (TBI) is increasingly appreciated to be highly prevalent and deleterious to neurological function 1,2

The ability of these cells to persist in a quiescent state, evade apoptosis, and promote tumor angiogenesis underscores the need for targeted strategies

Regulating cellular iron homeostasis after SCI by chelating excess iron ions and modulating iron transport pathways can promote the differentiation of neural stem/progenitor cells into neurons and stimulate the regenerative potential of newborn neurons, accompanied by improvements in axonal reinnervation and myelin regeneration

Yuen WLP, Loh SYJ, Wang DB

doi: 10.3390/foods12214000 67 ManasaPSLKambleADChilakamarthiU