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The new oral CCl 4 -induced moderate liver damage model closely resembles the pathophysiological features seen in human ALF, which makes it a good model for testing possible therapeutic interventions [17, 18]

Glycogen accumulationhepatocytes not markedly enlarged

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Early on, the shifts tend to be modest: a quieter inflammatory tone, slightly less morning stiffness, better tolerance of activities that used to aggravate the issue

Recently, disrupted glycerophospholipid catabolism was reported in lysosomes of brain cells in Cln3 / mice [10]

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