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Antibiotics such as quinolones, beta-lactams and aminoglycosides are more effective against bacterial pathogens in vitro and in mouse models when combined with small compounds that block a bacterial enzyme involved in the formation of hydrogen sulfide [126]

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(1998) attempted to describe this factor by purifying a protein that selectively inhibited PLD2 and through sequencing and immunological analysis found that this protein was a mixture of - and -synucleins

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Using lipid nanoparticle-encapsulated siRNA to target and silence macrophage Cxcl2 markedly reduces myocardial neutrophil infiltration, diminishes inflammation, and reduces infarct size, concurrently demonstrating that aberrant macrophage chemotactic activity is a key target exacerbating post-inflammatory myocardial ischemic injury (33)

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