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This antioxidant has been associated with a delay in the clinical onset of the disease (Table 1) in transgenic mice that express mutant copies of the gene encoding SOD1 (an animal model of ALS) (Gurney et al., 1996)

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By promoting blood flow to injured areas, the peptide may accelerate the healing of tendons, ligaments, and muscles

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Three characteristic MAPK families (ERK, JNK and p38 kinases) have been identified in mammalian cells [47]