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The reversal of NET function results in efflux of cytoplasmic norepinephrine, normally contained in the , readily releasable pool 12) into the synaptic cleft, independent of calcium influx and the normal process of vesicular release (Raiteri and Levi, 1986

In particular, during ferroptosis, AECs collapse due to iron overload and lipid peroxidation-induced damage, releasing large amounts of ROS, which further activate fibroblasts and enhance ECM deposition, thereby accelerating pulmonary tissue stiffening (Cheng et al., 2021a)

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Impact of glutathione-S-transferase gene polymorphisms on enzyme activity, lung function and bronchial asthma susceptibility in Egyptian children