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10.1016/s1570-9639(03)00266-8 Biochim

Strategies to improve the EPR effect: a mechanistic perspective and clinical translation

At normal concentrations, hydrogen sulfide resulting from sulfur metabolism is cytoprotective, maintaining the integrity of the mucus layer

In this hypermetabolic state, the body releases a large number of reactive oxygen species (ROS), which can lead to thyroid epithelial cell damage, autoantigen activation of the immune system, and induction of autoimmune deterioration