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The mechanisms primarily include the activation of oxidative stress-related signalling pathways (such as NF-B and MAPK), oncogenes, inactivation of tumour suppressor genes, mutations in repair genes, and microsatellite instability [114,115,116]

To account for both hypertrophy of individual cells as well as possible increases in cell number, the percent areas of Iba-1 and GFAP staining as proportions of the annotated prefrontal cortex and cerebellum regions were quantified as previously published, 74 the Area quantification FL v2.3.4 module was used

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