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J Exp Clin Cancer Res (2018) 37:54

doi: 10.1038/s41589-022-01224-y 42 HalestrapA

As GSH acts as a major antioxidant in the brain, this impaired GSH redox status can lead to further oxidative damage and redox dysregulation, which is associated with cognitive deficits, neuronal death, and mortality in animal epilepsy models

Upon cellular uptake, ketone bodies are metabolized back to acetyl-CoA, which enters the TCA cycle to support ATP production via mitochondrial oxidative phosphorylation [36, 41] (Fig

Having validated that combined inhibition of GLUT1 and GCL synergistically induced cell death in xCT-high GBM and Ewings sarcoma cell lines, we next sought to identify other cancer types which might exhibit sensitivity to this drug combination

Additionally, TRIM16 overexpression enhances the expression of several antioxidant genes, including glutamatecysteine ligase catalytic subunit (GCLC), glutamatecysteine ligase modifier subunit (GCLM), HO-1, NADPH, and GPX2