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Nicotine can also acutely impair endothelial function, reducing nitric oxide availability and potentially contributing to oxidative stress and inflammation (9, 27, 28)

Although the etiology of pulmonary hypertension in smokers is not understood, recent work has suggested a role for inducible nitric oxide synthase (iNOS) in inducing oxidative stress, as explained in the study Pulmonary hypertension and vascular oxidative damage in cigarette smoke exposed eNOS(-/-) mice and human smokers, authored by Wright JL, Zhou S, Churg A, from the Department of Pathology, of the University of British Columbia, in Vancouver, Canada