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The drug-loaded nanoparticles demonstrated excellent biocompatibility, successfully crossed the bloodbrain barrier via intravenous injection without causing significant damage, and effectively inhibited A1-42 aggregation, making them a promising treatment for Alzheimer's disease[113]

Mitochondrial and synaptic abnormalities in ASD-related syndromes The growing body of evidence reveals that the ability of mitochondria to adapt to the changing environment and energy demand is impaired in various ASD-related syndromes [325, 326]