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This encompasses details such as drug names, active/historical use, frequency of use, risky needle-using behavior, visible signs of IDU, last use, skin popping, harm reduction interventions, and the existence of IDU
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(1985) described PVL as a disease with unclear etiology, but typically associated with tobacco use.2 However, the role of tobacco in PVL lesions is unknown since these lesions are seen in smokers and nonsmokers (Table 1).1,2,515 Several studies evaluated alcohol use by PVL patients, but the relation between them was not stablished (Table 1).1,9,11,13 In recent years, it has been hypothesized that human papillomavirus (HPV) may influence both potentially and already stablished oral malignant lesions.16 Although the association between oral squamous cell carcinoma (OSCC) and HPV is already mentioned, its influence on PVL cases is not confirmed yet.17 Over the last decades, some studies reported different and contradictory frequencies of HPV DNA detection in PVL (Table 2).1,7,10,18,19 About other possible etiologies, there are few studies with PVL that tried to identify the presence of Candida albicans
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