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Almost no PtCo@G@CPB was observed in the visceral organs, which also demonstrated that oral administration with PtCo@G@CPB had superior biocompatibility

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A loss of neuroprotective function is likely due to the genetic mutation causing HD which results in abnormal, dysfunctional astrocytes expressing the mHTT protein and the promotion of neuroinflammation as discussed above

5-MTHF is the active form of folate that can be absorbed through the diet or generated in the liver from folic acid

Animal studies show axonal regrowth, reduced swelling, stabilized neurotransmitters, and improved recovery after brain or spinal-cord injurymediated through vascular and neuronal repair pathways