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Human Mol Genet

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LAG-3 potentiates the survival of Mycobacterium tuberculosis in host phagocytes by modulating mitochondrial signaling in an in-vitro granuloma model

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Using HCV replicon systems and chimeric mice models infected with HCV genotypes 2a and 1b, PYC was shown to enhance the antiviral efficacy of ribavirin, IFN, and telaprevir, either synergistically or additively ( One key mechanism underlying PYCs antiviral activity is its ability to modulate oxidative stress