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The primary mechanism of action of paclitaxel involves microtubule stabilization, which disrupts cell division and induces apoptosis in rapidly proliferating cancer cells (Kaveh Zenjanab et al., 2024)

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When NAD+ levels drop, mitochondrial function suffers, energy production declines, and metabolism slows

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These findings indicated that upregulation of ACE2 activity may suppress abnormal proliferation of PASMCs and help reverse hypoxia-induced pulmonary vascular remodeling [151]

Granzyme B, reported to be expressed on activated killer T cells, the transcription factor Tox/Tox2, reported to be involved in T cell exhaustion, and the co-repressor receptor TIM-3 were found at sites of CD8+ T cell infiltration, whereas PD-1, LAG3, and TIGIT-positive cells were rarely observed in the tissue