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However, the comprehensive mechanisms remain to be elucidated

We find that the G198A mutant exhibits significantly lower O 2 consumption activity at all [CSSH] tested, in striking contrast to the G206A mutant, which is only modestly affected (Fig

Metabolic intermediates including -ketoglutarate and acetyl-CoA fulfill biosynthetic demands for nucleotide/lipid production while enabling epigenetic reprogramming via TET (Ten-eleven translocation enzymes) dioxygenase-mediated DNA demethylation (108, 109)