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Arutjunyan, A., et al

Rheumatology 62 , 12641271 (2023)

To attenuate the disruption to energy homeostasis in cells undergoing excessive autophagic flux, activation of the transcription factors TFEB and TFE3 promotes the expression of FAM134 family proteins and TEX264, and the regulation of casein kinase 2 (CK2) mediated via phosphorylation enhances the affinity of autophagy receptors for LC3 (Fig

[] These promising data come from the FGF19 mimetic phase II trial in the treatment of NASH patients and could open the door for a new drug for NASH patients

Benefits of iron chelators in the treatment of Parkinsons disease