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SPX proteins, as phosphorus receptors, can avoid toxicity caused by phosphorus over-accumulation by interacting with PHR1 ( Arabidopsis ) or PHR2 ( Oryza sativa ), and inhibiting their transcriptional activities under normal conditions, while SPX proteins do not affect PHR transcriptional activities when plants are in low phosphorus environments (Wang et al., 2014)

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Recent evidence suggests systemic therapy, either targeted agents or immunotherapy, can be effective in combating BrMs, especially those originating from well-characterized primary tumors ( The establishment of metastatic cancer encompasses entering the central nervous system (CNS) from the blood and crossing the BBB ( Recent accumulating evidence suggests that B cells play a multifaceted and relatively underexplored role in shaping the BrM tumor microenvironment (TME)