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379 The second possible mechanism is the autoantigen-like reaction triggered by the Atopobium antigen described in rheumatoid arthritis (RA)

showed that AGE-RAGE increased TGF- expression and secretion and inhibited stromal cell mineralization (105)

146 Previous studies in Japan, Italy, and the Sultanate of Oman have suggested that altered levels of adiponectin reflect impairments in social interaction, and may be implicated in the pathophysiology of autism

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427 Moreover, lung microbiome selectivity, such as cough, mucociliary clearance, and host immune defense mechanisms, is often impaired in critically ill patients as a result of both the disease itself and pharmacological interventions (e.g., sedation and corticosteroids)