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Systemic treatments with tyrosine kinase inhibitor and platinum-based chemotherapy in patients with unresectable or metastatic hepatocholangiocarcinoma

Under most physiological conditions, GSH buffers oxidants in the cytoplasm and mitochondria and supports nuclear repair processes, thereby increasing the threshold for regulated cell death and, in certain pathological settings, contributing to therapy resistance

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10.1042/bj1340707 11 BoverisA.CostaL

It is possible that CIT supplementation stimulated the production of NO and CIT, but the effect on FMD was not evident in the absence of endothelial dysfunction

Sections were stained with periodic acid-Schiff (PAS) stain as described ( BRONCHOALVEOLAR LAVAGE Mouse lungs were inflated with 500 L of PBS and centrifuged to sediment cells