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These findings reveal the molecular basis of manganese homeostasis and provide a structural blueprint for understanding and potentially treating Mn 2+ -related disorders

and these chain reactions persist at membrane interfaces until terminated by antioxidants or substrate depletion, demonstrating the efficiency of radical-mediated lipid damage within the confined region (Fig

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Copper accumulation: While copper is essential, excessive accumulation from multiple sources could theoretically lead to oxidative stress and uniform inflammation, distinct from localized purging

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