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Thus, preserving adipocyte CI function with NDI1 sustained NADH oxidation and mitochondrial respiration, enabling visceral WAT to remodel through hyperplasia rather than hypertrophy during HFD feeding

After that, at 24 min, 100%/0% was kept until 27 min

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It's whether the experimental design isolates each compound's contribution, controls for confounding variables, and monitors the right endpoints

doi: 10.3390/ijms26136456, 162 WuQ.WangW.HuangZ.LinX.YaoM.CaiC.et al