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While GLP-1 and GIP receptor agonists are highly effective for initiating caloric deficits, human metabolism is highly adaptive

In the erlotinib-treated HT22 hippocampal neuron model, CHY enhanced GPX4 expression, reduced ACSL4, suppressed ROS, and improved cell viability, further supporting its role in ferroptosis regulation (Shang JF

Scavenging ROS with N-acetylcysteine inhibits insulin secretion by decreasing ROS (Figure 6A)

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