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Role of alpha-7-nicotinic acetylcholine receptor in Alzheimers disease

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Excessive mtROS production or impaired antioxidant defenses disrupts redox homeostasis and mitochondrial dynamics [87], leading to mtDNA damage and impaired mitophagy, thereby amplifying mitochondrial dysfunction [88, 89]

At the same time, TB-500 helps regulate inflammation and supports cellular repair

Additionally, hyperinsulinemia driven by pancreatic hypersecretion and/or impairments in hepatic insulin clearance could explain changes in glomerular filtration rates and increased renal gluconeogenesis, which in turn causes fatty liver deposition and, as a direct consequence, may lead to dysglycemia (Naderpoor et al., 2017