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Myocardial infarction accelerates atherosclerosis

Samples or metabolites with more than 30% missing values (n = 8) were removed

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Consequently, most genetic contributions to neurodegenerative phenotypes escape the strict pathogenic/benign dichotomy defined by ACMG, creating a bioinformatic challenge: common variants of small effectand even variants classified as benignmay exert additive influences on phenotype

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