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Th17-associated inflammation usually contributes to the neutrophilic phenotype asthma, which is often characterized by greater severity, airflow obstruction, and steroid resistance (52)

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These changes are directly responsible for the phenomenon of fetal immunotolerance, inhibiting the development and activation of a subpopulation of T lymphocytes that can stimulate inflammatory reactions within the uterine endometrium, while maintaining the endometrial and trophoblast defense against pathogens (165)