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Scientific reports have stated two possible pathophysiological mechanisms: (i) severe acute respiratory syndromecoronavirus-2 interaction with hemoglobin molecule, through CD26, CD147, and other receptors located on erythrocyte and/or blood cell precursors

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The bioactive compounds in H

Calcium homeostasis abnormalities can lead to mitochondrial malfunction, oxidative stress, and cytotoxicity [121]

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