slc25a39 glutathione DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Dual regulation of SLC25A39 by
Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis ScienceDirect SLC25A39 overexpression exacerbates lung adenocarcinoma progression and is negatively regulated by AFG3L2 npj Precision Oncology FGF21 reduced the degradation of SLC25A39 protein and facilitated the Download Scientific Diagram SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Nature glutathione and mitochondrial function SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation
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