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Modulation of basal and corticotropin-releasing factor-stimulated proopiomelanocortin gene expression by vasopressin in rat anterior pituitary

This binding triggers membrane pore formation, leading to cell lysis and death, leaving normal cells unaffected (Goldberg et al., 2010)

Form:Lyophilised Solid

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[2] Nitric Oxide System & Endothelial Research Research applications include studies of: Endothelial nitric oxide synthase (eNOS) modulation through upstream kinase pathways Caveolin-1 (Cav-1) binding interactions with eNOS in cell culture Nitric oxide (NO) production mechanisms and microcirculatory effects in vascular models Platelet aggregation pathways in coagulation research Studies examine NO production mechanisms following cav-1 displacement in endothelial cell models and microcirculatory perfusion in preclinical research systems

Molecular and clinical aspects of GHRH receptor mutations