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Besides an increased homocysteine export from tissues into plasma that may be regulated by a reduced-homocysteine carrier (Blom, 2000), hyperhomocysteinemia can result from reduced renal function, which may have a central role in plasma homocysteine clearance, and from chronic hepatic insufficiency

doi:10.1007/s11011-012-9324-8, PMID: 22718491 Morris G, Maes M (2012) Increased nuclear factor-B and loss of p53 are key mechanisms in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS)

The main advantage of these systems is a direct contact between the barrier cells (Nzou et al., 2018

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In rodent models of IBD, the antioxidant potential of RSV has been investigated

Lactate appears to modulate cardiomyocyte responses by enhancing protein lactylation and YTHDF2 expression, suggesting that reducing lactylation could be a promising therapeutic strategy for MI/RI [241]