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It has been demonstrated that administration of GluN2B-specific inhibitors, ifenprodil and conantokin-G, ameliorates ischemia-mediated infarct size and neuronal death by decreasing neurological deficits, restoring cytoarchitecture, and increasing the expression of pro-survival CREB molecules 69,70

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In the same study, this ITPA genotype was associated with clinical effectiveness [62]