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Description

10.1136/jmedgenet-2015-102986 43 GalaskoD

Molecular mechanisms underlying MNPs-induced cardiovascular toxicity MNPs trigger cardiovascular toxicity through interconnected mechanistic pathways

doi: 10.1074/jbc.M610363200

Results Data was obtained for cohorts of nonexercise and postexercise ME/CFS and sedentary control (SC) subjects (Table 1)

with modifications by Alice Callahan

6-Phosphogluconolactonase then hydrolyzes the lactone to 6-phosphogluconate via a proton-relay mechanism involving conserved histidine and aspartate residues