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According to Hernandez-Perez et al

Dihexa binds to HGF and potentiates its activity at the c-Met receptor on neurons

The treatment of PFIC has long been based on the use of drugs, such as ursodeoxycholic acid (UDCA), rifampicin, and cholestyramine, or surgical biliary diversion (SBD), aimed at reducing symptoms, especially pruritus [13]

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2d,e), whereas empty-vector control cells were not sensitized to DNA-damaging agents by TMLHE knockdown or mildronate (Extended Data Fig

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