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In summary, the pathological mechanisms of sepsis-associated ARDS are not limited to injury of a single cell type but involve complex intercellular network communication mediated by multi-source exosomes

p53 is required for brown adipogenic differentiation and has a protective role against diet-induced obesity

demonstrated that Glutathione loss and a high level of oxidative stress have been shown to promote crystallin aggregation and damage lens transparency, thus increasing the cataract formation rate 10

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For instance, propionic acidemia/aciduria (PA) is a rare and fatal genetic disease