increased glutathione levels gliomas autophagy

increased glutathione levels gliomas autophagy and Its Metabolic Enzymes in Gliomal Tumor Tissue and the Peritumoral Zone at Different Degrees of Anaplasia Nanotechnology as a new strategy

Name: increased glutathione levels gliomas autophagy and Its Metabolic Enzymes in Gliomal Tumor Tissue and the Peritumoral Zone at Different Degrees of Anaplasia Nanotechnology as a new strategy
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Nanotechnology as a new strategy for the diagnosis and treatment of gliomas Glutathione metabolism related gene signature predicts prognosis and treatment response in low grade glioma Aging The GlutamateGlutathione axis in neuropsychiatric disorders and cancer: From shared mechanisms to non invasive biomarkers ScienceDirect Revisiting the potential of regulated cell death in glioma treatment: a focus on autophagy dependent cell death, anoikis, ferroptosis, cuproptosis, pyroptosis, immunogenic cell death, and the crosstalk between them PMC Targeting ferroptosis opens new avenues in gliomas The dual role of autophagy in cancer stem cells: implications for tumor progression and therapy resistance Journal of Translational Medicine Springer Nature Link

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[DOI] [PubMed] [Google Scholar] 149.Ghosh S., Chakraborty N., Banerjee A., Chatterjee T., Acharya K

The first indication that TRIMs as a family could directly intersect with the autophagy machinery was published in 2014, when TRIM5, TRIM6, TRIM17, TRIM22, and TRIM49 were shown to interact with the autophagy regulators ULK1 and Beclin 1 (Mandell et al., 2014)