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In the case of AD, however, the persistence of autophagy induction in the face of growing lysosome failure becomes counterproductive, leading to a uniquely massive autolysosome accumulation in the most compromised neurons (Fig

Overexcitation of the neuron leads to depolarization of the presynaptic membrane, prompting the opening of voltage-gated Ca 2+ channels and allowing a massive influx of Ca 2+ into the presynaptic neuron [13,14,15]

Subsequently, protein was eluted by linearly increasing the concentration of HS Buffer to 50%

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UDP, uridine diphosphate

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