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Under basal physiological conditions, Nrf2 is sequestered in the cytoplasm through its interaction with Kelch-like ECH-associated protein 1 (Keap1), which facilitates Nrf2 ubiquitination and subsequent proteasomal degradation (Olagnier et al., 2020)

These interconnected mechanisms are further summarized in Figure 3, which highlights the convergence of oxidative stress and epigenetic dysregulation in toxin-induced carcinogenesis

Both animal and human studies indicate that MOTS-c levels tend to decline with advancing age in tissues like skeletal muscle and in circulation

The study further illustrates the role of digital platforms in the gentrification process in the information era and proposes strategies for addressing its challenges

Such biomarkers would be invaluable for selecting appropriate candidates for clinical trials and for monitoring treatment responses in real time (180)