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[3] [9] The mechanism of action involves binding to both GIP and GLP-1 receptors, which are naturally occurring incretin hormones

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[3] Hyperpigmentation in B12 deficiency occurs as a result of (1) decreased levels of reduced glutathione which activates tyrosinase and results in increased melanin synthesis, (2) defect in transfer of melanin from melanocytes to keratinocytes resulting in pigment incontinence, (3) increased biopterin in B12 deficiency increases hydroxylated phenylalanine which results in hyperpigmentation

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MOTS-C has been shown to reverse these age-related metabolic changes by improving insulin sensitivity and promoting cellular energy production

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