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Transl Psychiatry (2015) 5(9):e648

Summary of laboratory model and mechanistic signaling observations Data from documented laboratory peptide signaling studies Documented interactions in GH secretagogue research models Peak elevation in research models Teichman et al., 2006 Reduced time to peak GH pulse Bowers et al., 1991 Physiologic rhythm maintenance Teichman et al., 2006 Verified in laboratory settings Raun et al., 1998 GH secretagogue research suggests pathway activity that supports protein synthesis and lipid oxidation modeling

This modulation ultimately leads to a suppression of the production and secretion of inflammatory cytokines, including TNF-, IL-1, and IL-6, thereby regulating inflammatory reactions and mitigating tissue damage in T cell-mediated autoimmune diseases, such as RA, MS, and inflammatory bowel disease (218)