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AMPK inhibits ULK1-dependent autophagosome formation and lysosomal acidification via distinct mechanisms

For instance, atrophic gastritis reduces stomach acid necessary for releasing B from food proteins

N-3 PUFAs activate Hippo, promote yes-associated protein 1 exocytosis, weaken cross-talk between yes-associated protein 1 and Nrf2, and ultimately activate ferroptosis sensitivity in ovarian GCs

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Similar trend was observed in CAL-62 cells

CKD contributes to atherosclerosis not only by increasing traditional risk factors like hypertension (Valdivielso et al., 2019), dyslipidemia and inflammation, but also through CKD-specific alterations like albuminuria and the accumulation of uremic retention solutes (Vondenhoff et al., 2024)