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Therefore, oxidative cell death mechanisms offer the potential to selectively target and eliminate cancer cells while sparing healthy tissues, thereby minimizing the adverse effects commonly associated with traditional therapies

32, 1986 (2025), Abstract RIPK1 autophosphorylation at S161 mediates cell death and inflammation.: Koerner, L., Li, X., et al

Zhang G, Xu M, Zhang C, Jia H, Zhang H, He M (2021) Comparative transcriptomic and expression profiles between the foot muscle and mantle tissues in the giant triton snail Charonia tritonis

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For example, the PRKN and PINK1 genes mediate mitophagy (Pickrell and Youle, 2015) and are the major causes of autosomal recessive early onset mPD (Supplementary Table 1)