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Corticosteroids suppress bone formation and promote bone resorption

This surprises people who have read about it in the context of fat loss and assumed it would work the same way GLP-1 medications do

The other pathway is non-enzymatic autoxidation, wherein labile Fe 2+ catalyzes the generation of ROS through Fenton chemistry, initiating peroxidation of PUFA chains Regardless of the initiating route, these oxidation events trigger a self-propagating lipid radical chain reaction, which, if not mitigated by GPX4, leads to membrane disruption and ferroptotic cell death

J Allergy Clin Immunol (2022) S0091-6749(22):011824

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Rev Bras Zootec 39:348363