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Emerging evidence suggests that microglia, as key neuroimmune modulators within the CNS, influence the incidence and progression of cardiovascular diseases, including hypertension, myocardial infarction, and ischemia/reperfusion injury, through mechanisms potentially linked to altered autonomic nervous system activity (Wang M

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One of the most well-established properties of GSH is its role in maintaining redox homeostasis, but it is also emerging as a mediator of redox signaling through mechanisms such as protein S-glutathionylation and/or GSNO-mediated protein S-nitrosylation (Zhang and Forman, 2012)

This suggests that CU may exert therapeutic effects on PD through its anti-inflammatory and antioxidant properties in neuronal cells (Yu et al., 2016)